Page 14 - APN1022book
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  Labor getting bad agriculture advice
THE Nationals leader David Littleproud said recent revelations that a senior agriculture policy advisor for Prime Min- ister Anthony Albanese is a former GetUp cam- paigner would send a shiver down the spine of regional farmers across Australia.
mainstream farming “in- herently cruel” is deeply concerning and shows Mr Albanese is prioritising activists over farmers.
production systems or its success.”
industry was far from de- clining but needed sup- port and practical solu- tions to ensure its future success.
Mr Littleproud said the fact the senior agriculture adviser previously called
“It is obvious Mr Al- banese and Agriculture Minister Murray Watt do not believe in agriculture
Mr Littleproud added the bad agriculture advice was especially worrying for the live sheep export trade.
“The Nationals are calling for common sense.
“Today’s claim proves Mr Albanese is getting bad advice when it comes to our crucial agriculture industry,” Mr Littleproud said.
“They should be fighting for our farming industry, not employing people who seek to de- stroy it.”
“A r o u n d 3 0 0 0 l i v e l i - hoods are at risk by this government’s decision to ban the live sheep export trade in WA,” Mr Little- proud said.
 The Nationals leader David Littleproud weighs in on recent revelations.
The live sheep export trade is worth $92 million a year.
“A n y p l a n t o f u r t h e r harm the trade would disrupt global food secu- rity as well as Australian jobs.”
“Agriculture Minister Senator Watt previously said the science showed the live sheep export trade should be banned,” Mr Littleproud said.
“This isn’t just about our export trade but also food security and if Labor ban live sheep, cattle is next.
“It’s now beholden on the Prime Minister to make public the science the Agriculture Minister said was behind their de- cision to ban live sheep, because the industry de- serves to see it.”
“The previous coalition government increased regulations for sheep exporters to ensure they were the absolute best in the world.
Mr Littleproud said the
 The virus is present in both nasal and oral secretions of infected animals and can be detected even before its appearance in blood and clinical signs.
Role of different matrices for secondary spread of ASF
THE following are the different matrices for the secondary spread of Af- rican swine fever. Oral-nasal excretions and secretions
The virus is present in both nasal and oral secre- tions of infected animals and can be detected even before its appearance in blood and clinical signs.
tiate a new transmission cycle when the defrosted carcasses are visited the following spring by sus- ceptible wild boars.
The quantity of shed virus is relatively low, though sufficient to trigger new infections.
In the natural history of ASF in the wild boar cycle, the virus survival in carcasses plays a cru- cial role.
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  In the oral-nasal fluids, the virus is shed for a fewdays–twotofour– while its half-life is not known.
It outlives its host.
Oral and nasal fluids are likely to be involved in the direct contact spread of the infection. Blood
Once an infected wild boar dies, the virus re- mains infectious in the carcass for an extended period of time.
The virus is detected in the blood of infected wild boars at two to five days – average is three days – post exposure.
In such an epidemio- logical framework, safe removal of carcasses from the environment and their disposal is one of the most important disease control meas- ures, without which ASF eradication from wild boar populations is not possible.
The detection of the virus in the blood is con- comitant with the onset of clinical signs.
Offal
The virus is massively shed in the blood where it can survive for 15 weeks at room tempera- ture, months at 4C and indefinitely when frozen.
The virus survival rates in offal are similar to those in carcasses.
 Page 14 – Australian Pork Newspaper, October 2022
A frozen carcass can maintain infectious virus for months, which means that the pathogen can overwinter even in the temporary absence of any live host and ini-
* continued P15 www.porknews.com.au
The blood contami- nation of soil, hunting premises and tools, in- cluding knives, clothes and cars used for trans- port of infected hunted animals are important sources for the local persistence and further spread of the virus.
Whenever an infected animal is dressed in the field, the offal – in- cluding viscera, skin, head and other parts of the body – becomes an important potential source of the virus.
 Raw meat
Particularly in winter, when hunting activities take place, offal that has not been properly dis- posed of has the poten- tial to increase the risk of secondary infections and the spread of the disease. Faeces and urine
The virus is present in the meat of sick animals too.
Both are infectious and the half-life of the virus is determined by the environmental tem- perature.
Since the virus is re- sistant to putrefaction, it can survive for more than three months in meat and offal.
ASFV survives longer in urine than in faeces.
It remains infective for almost one year in dry meat and fat, and it survives indefinitely in frozen meat.
Its half-life in urine ranges from 15 days at 4Cto3daysat21C.
Also, the meat rep- resents an important source for both the local maintenance and pos- sible further spread of the virus.
In faeces, virus half- life ranges from eight days at 4C to five days at 21C and the virus DNA is detectable from two to four years.
Frozen meat of infected wild boars can ensure survival of the virus for years and thus represents a possible source for new epidemics.
The half-life of the virus is strongly affected by enzymes – proteases and lipases – produced by bacteria colonising faeces and urine.
Carcasses
Thus, the exact survival time in the forest where ASF is actively circu- lating is not fully com- parable to the estimates obtained in laboratory conditions.
As in meat, the virus can survive in whole car- casses for a very long time depending on am- bient temperatures.
However, in areas highly contaminated by infected faeces and urine, the risk of sec- ondary spread of the virus will be more likely
 





























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